How Urolithin A Drives Mitochondrial Renewal and Slows Immune Aging
Story at-a-glance
- As you age, your thymus produces fewer newly formed cells responsible for responding to unfamiliar pathogens, reducing your immune system's adaptability. This shift, combined with persistent inflammation, defines the core features of immune aging
- Urolithin A, a postbiotic compound, stimulates mitochondrial renewal in aging immune cells. A recent study shows it can increase naïve-like T cells and strengthen immune surveillance in just four weeks of supplementation
- Clinical findings show that urolithin A boosts mitochondrial renewal pathways, increases markers linked to mitochondrial biogenesis, and improves immune cell metabolism
- Beyond immune health, studies reveal that urolithin A influences cancer pathways, enhances muscle strength and endurance, improves fatty liver markers, and reshapes metabolic signaling involved in obesity and insulin resistance
- Beyond using urolithin A, you can also support your mitochondria by lowering linoleic acid intake, eating the right carbohydrates, limiting environmental toxins, and supporting NAD⁺ production with niacinamide.
- Most people tend to think of immune health mainly as a defense against seasonal colds or infections. In reality, its influence runs deeper. A well-balanced immune system helps regulate inflammation, maintain internal stability, and support energy and vitality. As the years pass, however, the immune system undergoes natural changes that are part of the aging process itself.1
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